The investigators plan to study the etiology of vulvodynia, a condition characterized by chronic vulvar discomfort, most often described as burning pain, occurring in the absence of relevant visible findings or a specific, clinically identifiable, neurologic disorder. Our hypothesis is that vulvodynia is the result of an altered immuno-inflammatory response mechanism that occurs as a consequence of reproductive, gynecologic, environmental, or psychological exposures, with abnormal vaginal microflora and genetic polymorphisms as potential modifiers of the effects of interest. Therefore, the investigators propose to collect information about reproductive, gynecological and environmental exposures, psychological trauma and psychiatric morbidity, and biological markers of immuno-inflammation and nerve fiber proliferation.
Age range
18 Years – 40 Years
Sex
FEMALE
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Reproductive, gynecological and environmental exposures and their association with vulvodynia & immuno-inflammatory response
Timeframe: Baseline
Past psychological trauma and psychiatric morbidity and their association with vulvodynia & immuno-inflammatory response
Timeframe: 6 month post baseline visit
immuno-inflammation and nerve fiber proliferation and vulvodynia.
Timeframe: Baseline